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Download for free: *******bit.ly/MolBioCell5thPDFFree In Molecular Biology of the Cell 5th Edition PDF, new material has been included on many topics, ranging from epigenetics, histone modifications, small RNAs, and comparative genomics, to genetic noise, cytoskeletal dynamics, cell-cycle control, apoptosis, stem cells, and novel cancer therapies. This book gives readers a conceptual framework for the mass of information that we now have about cells. Release information: Genre: Biological Science Type: PDF Release: November 16, 2007. Language: English Pages: 1725 (in PDF) Size: 220 MB Authors: Bruce Alberts, Alexander Johnson, Julian Lewis, Martin Raff, Keith Roberts, Peter Walter =========================================== Download for free: *******bit.ly/MolBioCell5thPDFFree =========================================== Molecular Biology of the Cell 5th Edition PDF Ebook Free Download Additional tags: alberts cell biology 5th edition alberts molecular biology of the cell 5th edition biology of the cell alberts molecular biology of the cell 5th edition Molecular Biology of the Cell 5th Edition download molecular biology of the cell 5th edition pdf Molecular Biology of the Cell 5th Edition pdf download molecular biology of the cell by bruce alberts molecular biology of the cell ebook Molecular Biology of the Cell fifth Molecular Biology of the Cell fifth edition molecular biology of the cell pdf the molecular biology of the cell 5th edition
11 Jun 2013
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Download for free: *******bit.ly/MolBioCell5thPDFFree In Molecular Biology of the Cell 5th Edition PDF, new material has been included on many topics, ranging from epigenetics, histone modifications, small RNAs, and comparative genomics, to genetic noise, cytoskeletal dynamics, cell-cycle control, apoptosis, stem cells, and novel cancer therapies. This book gives readers a conceptual framework for the mass of information that we now have about cells. Release information: Genre: Biological Science Type: PDF Release: November 16, 2007. Language: English Pages: 1725 (in PDF) Size: 220 MB Authors: Bruce Alberts, Alexander Johnson, Julian Lewis, Martin Raff, Keith Roberts, Peter Walter =========================================== Download for free: *******bit.ly/MolBioCell5thPDFFree =========================================== Molecular Biology of the Cell 5th Edition by Alberts PDF Download Additional tags: alberts cell biology 5th edition alberts molecular biology of the cell 5th edition biology of the cell alberts molecular biology of the cell 5th edition Molecular Biology of the Cell 5th Edition download molecular biology of the cell 5th edition pdf Molecular Biology of the Cell 5th Edition pdf download molecular biology of the cell by bruce alberts molecular biology of the cell ebook Molecular Biology of the Cell fifth Molecular Biology of the Cell fifth edition molecular biology of the cell pdf the molecular biology of the cell 5th edition
11 Jun 2013
1981
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0:30
The molecular biology enzymes and kits & reagents market is projected to reach USD 12.69 Billion by 2021 from USD 5.77 Billion in 2016, growing at a CAGR of 17.1% during the forecast period of 2016 to 2021.
14 Sep 2016
221
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10:22
Entire Durston video: *******vimeo****/1775160 Astonishing DNA complexity demolishes neo-Darwinism Alex Williams excerpt of paper: The astonishing complexity of the dynamic information storage capacity of the DNA/chromosome system is, in itself, a marvel of engineering design. Such a magnificent solution to such a monster logistics problem could surely only come from a Master Designer. But the nature of the majority of this information poses an impossible conundrum for neo-Darwinists. *******creation****/images/pdfs/tj/j21_3/j21_3_111-117.pdf The Capabilities of Chaos and Complexity: David L. Abel - Null Hypothesis For Information Generation - 2009 To focus the scientific communitys attention on its own tendencies toward overzealous metaphysical imagination bordering on wish-fulfillment, we propose the following readily falsifiable null hypothesis, and invite rigorous experimental attempts to falsify it: Physicodynamics cannot spontaneously traverse The Cybernetic Cut: physicodynamics alone cannot organize itself into formally functional systems requiring algorithmic optimization, computational halting, and circuit integration. *******www.mdpi****/1422-0067/10/1/247/pdf *******mdpi****/1422-0067/10/1/247/ag Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/
27 Sep 2010
485
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9:57
The Capabilities of Chaos and Complexity: David L. Abel - Null Hypothesis For Information Generation - 2009 To focus the scientific community’s attention on its own tendencies toward overzealous metaphysical imagination bordering on “wish-fulfillment,” we propose the following readily falsifiable null hypothesis, and invite rigorous experimental attempts to falsify it: "Physicodynamics cannot spontaneously traverse The Cybernetic Cut: physicodynamics alone cannot organize itself into formally functional systems requiring algorithmic optimization, computational halting, and circuit integration." A single exception of non trivial, unaided spontaneous optimization of formal function by truly natural process would falsify this null hypothesis. *******www.mdpi****/1422-0067/10/1/247/pdf *******mdpi****/1422-0067/10/1/247/ag Signature in the Cell - Book Review - Ken Peterson Excerpt: the “simplest extant cell, Mycoplasma genitalium — a tiny bacterium that inhabits the human urinary tract — requires ‘only’ 482 proteins to perform its necessary functions…(562,000 bases of DNA…to assemble those proteins).” ,,, amino acids have to congregate in a definite specified sequence in order to make something that “works.” First of all they have to form a “peptide” bond and this seems to only happen about half the time in experiments. Thus, the probability of building a chain of 150 amino acids containing only peptide links is about one chance in 10 to the 45th power. In addition, another requirement for living things is that the amino acids must be the “left-handed” version. But in “abiotic amino-acid production” the right- and left-handed versions are equally created. Thus, to have only left-handed, only peptide bonds between amino acids in a chain of 150 would be about one chance in 10 to the 90th. Moreover, in order to create a functioning protein the “amino acids, like letters in a meaningful sentence, must link up in functionally specified sequential arrangements.” It turns out that the probability for this is about one in 10 to the 74th. Thus, the probability of one functional protein of 150 amino acids forming by random chance is (1 in) 10 to the 164th. If we assume some minimally complex cell requires 250 different proteins then the probability of this arrangement happening purely by chance is one in 10 to the 164th multiplied by itself 250 times or one in 10 to the 41,000th power. *******www.spectrummagazine****/reviews/book_reviews/2009/10/06/signature_cell Intelligent Design: Required by Biological Life? K.D. Kalinsky - Pg. 11 Excerpt: It is estimated that the simplest life form would require at least 382 protein-coding genes. Using our estimate in Case Four of 700 bits of functional information required for the average protein, we obtain an estimate of about 267,000 bits for the simplest life form. Again, this is well above Inat and it is about 10^80,000 times more likely that ID (Intelligent Design) could produce the minimal genome than mindless natural processes. *******www.newscholars****/papers/ID%20Web%20Article.pdf Could Chance Arrange the Code for (Just) One Gene? "our minds cannot grasp such an extremely small probability as that involved in the accidental arranging of even one gene (10^-236)." *******www.creationsafaris****/epoi_c10.htm Estimating the prevalence of protein sequences adopting functional enzyme folds: Doug Axe: Excerpt: Starting with a weakly functional sequence carrying this signature, clusters of ten side-chains within the fold are replaced randomly, within the boundaries of the signature, and tested for function. The prevalence of low-level function in four such experiments indicates that roughly one in 10^64 signature-consistent sequences forms a working domain. Combined with the estimated prevalence of plausible hydropathic patterns (for any fold) and of relevant folds for particular functions, this implies the overall prevalence of sequences performing a specific function by any domain-sized fold may be as low as 1 in 10^77, adding to the body of evidence that functional folds require highly extraordinary sequences. *******www.ncbi.nlm.nih.gov/pubmed/15321723 Evolution vs. Functional Proteins - Doug Axe - Video *******www.metacafe****/watch/4018222/evolution_vs_functional_proteins_where_did_the_information_come_from_doug_axe_stephen_meyer/ Axe Diagram for finding a functional protein domain out of all sequence space: The y-axis can be seen as representing enzyme activity, and the x-axis represents all possible amino acid sequences. Enzymes sit at the peak of their fitness landscapes (Point A). There are extremely high levels of complex and specified information in proteins--informational sequences which point to intelligent design. *******www.evolutionnews****/axediagram.jpg Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/
22 Mar 2010
900
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8:31
"Charles Darwin said (paraphrase), 'If anyone could find anything that could not be had through a number of slight, successive, modifications, my theory would absolutely break down.' Well that condition has been met time and time again. Basically every gene, every protein fold. There is nothing of significance that we can show that can be had in a gradualist way. It's a mirage. None of it happens that way. - Doug Axe PhD. Experimental Evolution in Fruit Flies - October 2010 Excerpt: "This research really upends the dominant paradigm about how species evolve".,,, as stated in regards to the 35 year experimental failure to fixate a single beneficial mutation within fruit flies. *******www.arn****/blogs/index.php/literature/2010/10/07/experimental_evolution_in_fruit_flies Waiting Longer for Two Mutations - Michael J. Behe Excerpt: Citing malaria literature sources (White 2004) I had noted that the de novo appearance of chloroquine resistance in Plasmodium falciparum was an event of probability of 1 in 10^20. I then wrote that ‘‘for humans to achieve a mutation like this by chance, we would have to wait 100 million times 10 million years’’ (Behe 2007) (because that is the extrapolated time that it would take to produce 10^20 humans). Durrett and Schmidt (2008, p. 1507) retort that my number ‘‘is 5 million times larger than the calculation we have just given’’ using their model (which nonetheless "using their model" gives a prohibitively long waiting time of 216 million years). Their criticism compares apples to oranges. My figure of 10^20 is an empirical statistic from the literature; it is not, as their calculation is, a theoretical estimate from a population genetics model. *******www.discovery****/a/9461 Estimating the prevalence of protein sequences adopting functional enzyme folds: Doug Axe: Excerpt: Starting with a weakly functional sequence carrying this signature, clusters of ten side-chains within the fold are replaced randomly, within the boundaries of the signature, and tested for function. The prevalence of low-level function in four such experiments indicates that roughly one in 10^64 signature-consistent sequences forms a working domain. Combined with the estimated prevalence of plausible hydropathic patterns (for any fold) and of relevant folds for particular functions, this implies the overall prevalence of sequences performing a specific function by any domain-sized fold may be as low as 1 in 10^77, adding to the body of evidence that functional folds require highly extraordinary sequences. *******www.ncbi.nlm.nih.gov/pubmed/15321723 Evolution vs. Functional Proteins - Doug Axe - Video *******www.metacafe****/watch/4018222 Stephen Meyer - Functional Proteins And Information For Body Plans - video *******www.metacafe****/watch/4050681 This following video is a bit more clear for explaining exactly why mutations to the DNA do not control Body Plan morphogenesis, since the mutations are the ‘bottom rung of the ladder’ as far as the 'higher levels of the layered information’ of the cell are concerned: Stephen Meyer on Craig Venter, Complexity Of The Cell & Layered Information *******www.metacafe****/watch/4798685 "The evidence for the adaptive divergence of gene switches is still thin. The best case involves the loss of protective armor and spines in sticklebacks, both due to changes in regulatory elements. But these elements represent the loss of traits, rather than the origin of evolutionary novelties...We now know that Hox genes and other transcription factors have many roles besides inducing body pattern, and their overall function in development - let alone in evolution - remains murky." *******www.evolutionnews****/2010/06/scott_f_gilbert_developmental035931.html Here is a more thorough critique of evo-devo: Nature's "Gems": Microevolution Meets Microevolution - Casey Luskin - August 2010 *******www.evolutionnews****/2010/08/nature_gems_microevolution_mee037171.html#more Cortical Inheritance: The Crushing Critique Against Genetic Reductionism - Arthur Jones - video *******www.metacafe****/watch/4187488 Astonishingly, actual motors, which far surpass man-made motors in 'engineering parameters', are now being found inside 'simple cells'. Articles and Videos on Molecular Motors *******docs.google****/Doc?docid=0AYmaSrBPNEmGZGM4ejY3d3pfMzlkNjYydmRkZw&hl=en Michael Behe - Life Reeks Of Design - 2010 - video *******www.metacafe****/watch/5066181 And in spite of the fact of finding molecular motors permeating the simplest of bacterial life, there are no detailed Darwinian accounts for the evolution of even one such motor or system. "There are no detailed Darwinian accounts for the evolution of any fundamental biochemical or cellular system only a variety of wishful speculations. It is remarkable that Darwinism is accepted as a satisfactory explanation of such a vast subject." James Shapiro - Molecular Biologist The following expert doesn't even hide his very unscientific preconceived philosophical bias against intelligent design,,, ‘We should reject, as a matter of principle, the substitution of intelligent design for the dialogue of chance and necessity,,, Yet at the same time the same expert readily admits that neo-Darwinism has ZERO evidence for the chance and necessity of material processes producing any cellular system whatsoever,,, ,,,we must concede that there are presently no detailed Darwinian accounts of the evolution of any biochemical or cellular system, only a variety of wishful speculations.’ Franklin M. Harold,* 2001. The way of the cell: molecules, organisms and the order of life, Oxford University Press, New York, p. 205. Professor Emeritus of Biochemistry, Colorado State University, USA Michael Behe - No Scientific Literature For Evolution of Any Irreducibly Complex Molecular Machines *******www.metacafe****/watch/5302950/ “The response I have received from repeating Behe's claim about the evolutionary literature, which simply brings out the point being made implicitly by many others, such as Chris Dutton and so on, is that I obviously have not read the right books. There are, I am sure, evolutionists who have described how the transitions in question could have occurred.” And he continues, “When I ask in which books I can find these discussions, however, I either get no answer or else some titles that, upon examination, do not, in fact, contain the promised accounts. That such accounts exist seems to be something that is widely known, but I have yet to encounter anyone who knows where they exist.” David Ray Griffin - retired professor of philosophy of religion and theology Poly-Functional Complexity equals Poly-Constrained Complexity *******docs.google****/Doc?docid=0AYmaSrBPNEmGZGM4ejY3d3pfMjdoZmd2emZncQ DNA - Evolution Vs. Polyfuctionality - video *******www.metacafe****/watch/4614519 Bio-Complexity *******bio-complexity****/ojs/index.php/main/issue/view/24 Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/2009/10/intelligent-design-anthropic-hypothesis_19.html
15 Oct 2010
2038
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1:43
The Animators: Light Productions Video Services - Cambrian Animals From Darwin's Dillemma *******www.lightproductionsvideo****/Cambrian-Animals.html The Producers: Illustra Media *******illustramedia****/ Articles and Videos on Molecular Machines and Motors *******docs.google****/Doc?docid=0AYmaSrBPNEmGZGM4ejY3d3pfMzlkNjYydmRkZw&hl=en Stephen Meyer - Functional Proteins And Information For Body Plans - video *******www.metacafe****/watch/4050681 Michael Behe - Life Reeks Of Design - 2010 - video *******www.metacafe****/watch/5066181 And in spite of the fact of finding molecular machines and motors permeating the simplest of bacterial life, there are no detailed Darwinian accounts for the evolution of even one such motor or system. "There are no detailed Darwinian accounts for the evolution of any fundamental biochemical or cellular system only a variety of wishful speculations. It is remarkable that Darwinism is accepted as a satisfactory explanation of such a vast subject." James Shapiro - Molecular Biologist The following expert doesn't even hide his very unscientific preconceived philosophical bias against intelligent design,,, ‘We should reject, as a matter of principle, the substitution of intelligent design for the dialogue of chance and necessity,,, Yet at the same time the same expert readily admits that neo-Darwinism has ZERO evidence for the chance and necessity of material processes producing any cellular system whatsoever,,, ,,,we must concede that there are presently no detailed Darwinian accounts of the evolution of any biochemical or cellular system, only a variety of wishful speculations.’ Franklin M. Harold,* 2001. The way of the cell: molecules, organisms and the order of life, Oxford University Press, New York, p. 205. *Professor Emeritus of Biochemistry, Colorado State University, USA Michael Behe - No Scientific Literature For Evolution of Any Irreducibly Complex Molecular Machines *******www.metacafe****/watch/5302950/ “The response I have received from repeating Behe's claim about the evolutionary literature, which simply brings out the point being made implicitly by many others, such as Chris Dutton and so on, is that I obviously have not read the right books. There are, I am sure, evolutionists who have described how the transitions in question could have occurred.” And he continues, “When I ask in which books I can find these discussions, however, I either get no answer or else some titles that, upon examination, do not, in fact, contain the promised accounts. That such accounts exist seems to be something that is widely known, but I have yet to encounter anyone who knows where they exist.” David Ray Griffin - retired professor of philosophy of religion and theology What I find very persuasive, to the suggestion that the universe was designed with life in mind, is that physicists find many processes in a cell operate at the 'near optimal' capacities allowed in any physical system: William Bialek - Professor Of Physics - Princeton University: Excerpt: "A central theme in my research is an appreciation for how well things “work” in biological systems. It is, after all, some notion of functional behavior that distinguishes life from inanimate matter, and it is a challenge to quantify this functionality in a language that parallels our characterization of other physical systems. Strikingly, when we do this (and there are not so many cases where it has been done!), the performance of biological systems often approaches some limits set by basic physical principles. While it is popular to view biological mechanisms as an historical record of evolutionary and developmental compromises, these observations on functional performance point toward a very different view of life as having selected a set of near optimal mechanisms for its most crucial tasks.,,,The idea of performance near the physical limits crosses many levels of biological organization, from single molecules to cells to perception and learning in the brain,,,," *******www.princeton.edu/~wbialek/wbialek.html Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/2009/10/intelligent-design-anthropic-hypothesis_19.html
5 Feb 2011
2204
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26:00
Will we become defenseless against bacteria? Will bacteria always find a way to infect and even kill us? The emergence of antibiotic resistant strains of bacteria poses an enormous problem around the world. Scientists believe that the overuse of antibiotics is increasing the appearance of these pathogens. In the US, increasing casualties resulting from drug resistant staphylococcus infections received wide media attention. While antibiotics only work on bacterial infections, many patients and doctors regard antibiotics as a front-line form of treating any type of infection. Antibiotics are often prescribed because the specific pathogen that is causing an illness is often difficult to determine. In some cases they are used as a preventative measure. But is this the best defense? Are there ways to beat bacteria at their own game? On September 18, 2008 at the Koshland Science Museum in Washington, D.C., Dr. Stuart Levy, professor of Molecular Biology and Microbiology at Tufts University School of Medicine and Dr. Linda Tollefson, Assistant Commissioner for Science at the U.S. Food and Drug Administration, discussed how to optimize antibiotic use and how to minimize the emergence of drug resistant pathogens. In part 1 of this 3 part video series, Dr. Levy discusses the basics of microbial pathogens, bacteria, and antibiotic resistance. And, Dr. Tollefson outlines the various types and classes of antibiotic drugs, approved uses, and current levels of effectiveness. Dr. Levy is Professor of Molecular Biology and Microbiology at Tufts University School of Medicine where he is the Director of the Center for Adaptation Genetics and Drug Resistance. He directs research on mechanisms of bacterial antibiotic resistance. Stuart Levy is also Staff Physician at the Tufts Medical Center and he also serves as the president of The International Alliance for the Prudent Use of Antibiotics. Dr. Tollefson is Assistant Commissioner for Science at the U.S. Food and Drug Administration (FDA). She previously served as Deputy Director of the Center for Veterinary Medicine (CVM), where she led CVM's efforts to implement a risk-based approach to address antimicrobial resistance, fulfilling a 2001 Congressional mandate, and was instrumental in the founding of the National Antimicrobial Resistance Monitoring System for Enteric Bacteria. Tollefson also served as Chief of Epidemiology in the Center for Food Safety and Applied Nutrition where she successfully investigated numerous outbreaks of food borne disease and served as liaison to the Centers for Disease Control and Prevention. Parts 2 and 3 will be published over the coming weeks.
17 Oct 2008
358
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27:36
On September 18, 2008 at the Koshland Science Museum in Washington, D.C., Dr. Stuart Levy, professor of Molecular Biology and Microbiology at Tufts University School of Medicine and Dr. Linda Tollefson, Assistant Commissioner for Science at the U.S. Food and Drug Administration, discussed how to optimize antibiotic use and how to minimize the emergence of drug resistant pathogens. In part 2 of this 3 part video series, Dr. Levy discusses how antibiotic resistance develops, the development practices drug companies employ when producing antimicrobials, and how this process may change in the future. Dr. Tollefson outlines how the FDA is encouraging the development of antibiotics in an industry that is mostly focussed on manufacturing drugs for chronic illnesses. Dr. Levy is Professor of Molecular Biology and Microbiology at Tufts University School of Medicine where he is the Director of the Center for Adaptation Genetics and Drug Resistance. He directs research on mechanisms of bacterial antibiotic resistance. Stuart Levy is also Staff Physician at the Tufts Medical Center and he also serves as the president of The International Alliance for the Prudent Use of Antibiotics. Dr. Tollefson is Assistant Commissioner for Science at the U.S. Food and Drug Administration (FDA). She previously served as Deputy Director of the Center for Veterinary Medicine (CVM), where she led CVM's efforts to implement a risk-based approach to address antimicrobial resistance, fulfilling a 2001 Congressional mandate, and was instrumental in the founding of the National Antimicrobial Resistance Monitoring System for Enteric Bacteria. Tollefson also served as Chief of Epidemiology in the Center for Food Safety and Applied Nutrition where she successfully investigated numerous outbreaks of food borne disease and served as liaison to the Centers for Disease Control and Prevention. Part 3 will be published next week. To view part one visit the MicrobeWorld Video archives at www.microbeworld****/look/MicrobeWorldVideo.aspx
28 Mar 2011
420
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36:12
In the final episode of this 3 part video series on how to optimize antibiotic use and how to minimize the emergence of drug resistant pathogens, Dr. Linda Tollefson, Assistant Commissioner for Science at the U.S. Food and Drug Administration, goes in depth on the use of antimicrobial drugs in agriculture, their efficacy, and adverse human health consequences. Dr. Stuart Levy, professor of Molecular Biology and Microbiology at Tufts University School of Medicine, discusses policy, regulatory and funding issues around antibiotic resistance. Both Dr. Tollefson and Dr. Levy take a handful of questions from the audience. The series, "Antibiotics: Is a Strong Offense the Best Defense?" was filmed on September 18, 2008 at the Koshland Science Museum in Washington, D.C. Parts 1 and 2 can be found at www.microbeworld****.
8 Nov 2008
162
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5:11
Johnson & Johnson today announced that Axel Ullrich, Ph.D., director of the Department of Molecular Biology at the Max Planck Institute of Biochemistry in Germany, whose discoveries have led to novel cancer therapies including Herceptin (trastuzumab)*, is the winner of the 2009 Dr. Paul Janssen Award for Biomedical Research. An independent committee of world-renowned scientists selected Dr. Ullrich, who on September 8 will receive a $100,000 prize during a ceremony in Beerse, Belgium.To view Multimedia News Release, go to *******www.prnewswire****/mnr/pauljanssenaward/38987/
2 Jul 2009
111
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2:10
*******www.GetWealthywithJohn**** John Milligan ways to make money fast (ways to make money fast) “ways to make money fast” waystomakemoneyfast support at *******www.GetWealthyWithJohn**** 1. Make Money Online *******cashmultipliersystem**** Make $1200 every day. Quickest and easiest money maker guaranteed. Search Results 1. Original Ways To Make Money Fast- She Made $20000 in 3 Days by ... 16 Oct 2005 ... You're broke, the cupboard is bare. I sympathize with you though I don't really understand how you could be in this position with the ... *******searchwarp****/swa21070.htm - Similar 2. Need To Make Money Fast? 10 Quick and Easy Ways To Earn Extra ... Learn how to make money fast. Here are 10 free quick and easy ways to earn extra money online. *******www.affiliatewatcher****/need-to-make-money-fast-10-quick-and-easy-ways-to-earn-extra-money-online/ - Cached - Similar 3. How to Make Money Fast - wikiHow wikiHow article about How to Make Money Fast. ... Later, you can read up on long-term ways to make money. Otherwise, hurry up and follow these steps so you ... *******www.wikihow****/Make-Money-Fast - Cached - Similar 4. Six Ways to Make Fast Cash Whether you need money for books, some "walking around cash" or the fare for a ticket home, here are six creative ways you can earn some extra bucks. *******www.youngmoney****/money.../six-ways-to-make-fast-cash/ - Cached - Similar 5. Unusual Ways To Make Money Unusual ways to make money, from unusual businesses and jobs to treasure hunting and more. ... Ways To Make Money Fast · Save Money - More Funny Ways ... *******www.unusualwaystomakemoney****/ - Cached - Similar 6. Money - 4 ways to make money Money - Find out the best ways to make money. here are 4 examples... ... (4 Ways To Make Money) · (3 Astonishing Facts About Fast, Easy Money) ... *******www.spiritual****.au/articles/money.../money-dthorsen.htm - Cached - Similar 7. 25 Unusual Ways to Make Quick Money - WalletPop 1 most desperate way to make some quick cash, and only applies in this situation: you have some money, ... Easy Ways to Make Money Fast: Sperm Donation? ... *******www.walletpop****/.../25-ways-to-make-quick-money - Cached - Similar 8. Make Money Online Make money fast Make money Online at home Make money online at home using legitimate ways to make money online with a money Guru. ... options you can try for earning some fast money with your blogs. ... *******www.scopeformoney****/ - Cached - Similar 9. Good Ways to Make Money Fast — Moola Days When people want to make money, they want to make it as fast as possible in a timely manner. Who doesn't want to make money if it were as simple as one, ... *******www.mooladays****/2009/.../good-ways-to-make-money-fast/ - Cached - Similar 10. Easy ways to make money fast: Sperm donation? - Mixx As college students get ready for a new year of classes, there's a lot more to worry about than Shakespeare and molecular biology. *******www.mixx****/.../easy_ways_to_make_money_fast_sperm_donation
6 Feb 2010
481
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5:51
The Capabilities of Chaos and Complexity: David L. Abel - Null Hypothesis For Information Generation - 2009 To focus the scientific communities attention on its own tendencies toward overzealous metaphysical imagination bordering on wish-fulfillment, we propose the following readily falsifiable null hypothesis, and invite rigorous experimental attempts to falsify it: Physicodynamics cannot spontaneously traverse The Cybernetic Cut: physicodynamics alone cannot organize itself into formally functional systems requiring algorithmic optimization, computational halting, and circuit integration. *******www.mdpi****/1422-0067/10/1/247/pdf *******mdpi****/1422-0067/10/1/247/ag Testing the Biological Fitness of Antibiotic Resistant Bacteria - 2008 *******www.answersingenesis****/articles/aid/v2/n1/darwin-at-drugstore List Of Degraded Molecular Abilities Of Antibiotic Resistant Bacteria: *******www.trueorigin****/bacteria01.asp Mathematically Defining Functional Information In Molecular Biology - Kirk Durston - short video *******www.youtube****/watch?v=vUeCgTN7pOo William Dembski Is Interviewed By Casey Luskin About Conservation Of Information - Audio *******intelligentdesign.podomatic****/player/web/2009-10-15T13_14_01-07_00 Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/
9 Jan 2010
675
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6:42
The Capabilities of Chaos and Complexity: David L. Abel - Null Hypothesis For Information Generation - 2009 To focus the scientific community's attention on its own tendencies toward overzealous metaphysical imagination bordering on wish-fulfillment, we propose the following readily falsifiable null hypothesis, and invite rigorous experimental attempts to falsify it: Physicodynamics cannot spontaneously traverse The Cybernetic Cut: physicodynamics alone cannot organize itself into formally functional systems requiring algorithmic optimization, computational halting, and circuit integration. *******www.mdpi****/1422-0067/10/1/247/pdf *******mdpi****/1422-0067/10/1/247/ag "LIFES CONSERVATION LAW: Why Darwinian Evolution Cannot Create Biological Information": Excerpt: Though not denying Darwinian evolution or even limiting its role in the history of life, the Law of Conservation of Information shows that Darwinian evolution is inherently teleological. Moreover, it shows that this teleology can be measured in precise information-theoretic terms. (Dr. Dembski points out that the Conservation Law is clearly differentiated from the common definition of Theistic Evolution since mainstream Theistic evolutionists, such as Ken Miller and Francis Collins, hold that the Design found in life is not separable from the purely natural processes of Darwinian evolution, whereas Dembski and Marks are clearly saying the Design/Information found in life is detectable, can be separated from the natural processes we see in the universe, and "can be measured in precise information-theoretic terms".) *******www.evoinfo****/Publications/ConsInfo_NoN.pdf Mathematically Defining Functional Information In Molecular Biology - Kirk Durston - short video *******www.youtube****/watch?v=vUeCgTN7pOo Astonishing DNA complexity demolishes neo-Darwinism Alex Williams excerpt of paper: The astonishing complexity of the dynamic information storage capacity of the DNA/chromosome system is, in itself, a marvel of engineering design. Such a magnificent solution to such a monster logistics problem could surely only come from a Master Designer. But the nature of the majority of this information poses an impossible conundrum for neo-Darwinists. *******creation****/images/pdfs/tj/j21_3/j21_3_111-117.pdf Conservation of Information in Computer Search (COI) - William A. Dembski - Robert J. Marks II - Dec. 2009 Excerpt: COI puts to rest the inflated claims for the information generating power of evolutionary simulations such as Avida and ev. *******marksmannet****/RobertMarks/REPRINTS/2009_BernoullisPrinciple.pdf "... no operation performed by a computer can create new information." Douglas G. Robertson, "Algorithmic Information Theory, Free Will and the Turing Test," Complexity, Vol.3, #3 Jan/Feb 1999, pp. 25-34. The Evolutionary Informatics Lab: *******www.evoinfo****/index.html Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/
9 Jan 2010
391
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The first thought I had when I saw the bacteriophage virus is that it looks similar to the lunar lander of the Apollo program. The comparison is not without merit considering some of the relative distances to be traveled and the virus must somehow possess, as of yet unelucidated, orientation, guidance, docking, unloading, loading, etc... mechanisms. And please remember this level of complexity exists in a world that is far too small to be seen with the naked eye. This excellent video gives a small glimpse at the intricate, and humbling, complexity that goes into crafting the "simple" non-living bacteriophage virus. For anyone who has a molecular biology project they would like to bring to life with such stunning animation, please contact: Seyet LLC - Life Science Animation | Visual Communication Company *******www.seyet****/ "There are no detailed Darwinian accounts for the evolution of any fundamental biochemical or cellular system only a variety of wishful speculations. It is remarkable that Darwinism is accepted as a satisfactory explanation of such a vast subject." James Shapiro - Molecular Biologist CELL TO VIRUS - SIZE AND SCALE - Move Cursor At Bottom Of Graph To Zoom In *******learn.genetics.utah.edu/content/begin/cells/scale/ The Capabilities of Chaos and Complexity: David L. Abel - Null Hypothesis For Information Generation - 2009 To focus the scientific communitys attention on its own tendencies toward overzealous metaphysical imagination bordering on wish-fulfillment, we propose the following readily falsifiable null hypothesis, and invite rigorous experimental attempts to falsify it: "Physicodynamics cannot spontaneously traverse The Cybernetic Cut: physicodynamics alone cannot organize itself into formally functional systems requiring algorithmic optimization, computational halting, and circuit integration." A single exception of non trivial, unaided spontaneous optimization of formal function by truly natural process would falsify this null hypothesis. *******www.mdpi****/1422-0067/10/1/247/pdf *******mdpi****/1422-0067/10/1/247/ag Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/
20 Jan 2010
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"I have seen estimates of the incidence of the ratio of deleterious-to-beneficial mutations which range from one in one thousand up to one in one million. The best estimates seem to be one in one million (Gerrish and Lenski, 1998). The actual rate of beneficial mutations is so extremely low as to thwart any actual measurement (Bataillon, 2000, Elena et al, 1998). Therefore, I cannot ...accurately represent how rare such beneficial mutations really are." (J.C. Sanford; Genetic Entropy page 24) - 2005 Estimation of spontaneous genome-wide mutation rate parameters: whither beneficial mutations? (Thomas Bataillon) Abstract......It is argued that, although most if not all mutations detected in mutation accumulation experiments are deleterious, the question of the rate of favourable mutations (and their effects) is still a matter for debate. *******www.nature****/hdy/journal/v84/n5/full/6887270a.html Distribution of fitness effects caused by random insertion mutations in Escherichia coli Excerpt: At least 80% of the mutations had a significant negative effect on fitness, whereas none of the mutations had a significant positive effect. *******www.springerlink****/content/r37w1hrq5l0q3832/ High Frequency of Cryptic Deleterious Mutations in Caenorhabditis elegans ( Esther K. Davies, Andrew D. Peters, Peter D. Keightley) "In fitness assays, only about 4 percent of the deleterious mutations fixed in each line were detectable. The remaining 96 percent, though cryptic, are significant for mutation load...the presence of a large class of mildly deleterious mutations can never be ruled out. " *******www.sciencemag****/cgi/content/abstract/285/5434/1748 But in all the reading Ive done in the life-sciences literature, Ive never found a mutation that added information All point mutations that have been studied on the molecular level turn out to reduce the genetic information and not increase it. Lee Spetner - Ph.D. Physics - MIT - (Not By Chance: Shattering the Modern Theory of Evolution) "Bergman (2004) has studied the topic of beneficial mutations. Among other things, he did a simple literature search via Biological Abstracts and Medline. He found 453,732 mutation hits, but among these only 186 mentioned the word beneficial (about 4 in 10,000). When those 186 references were reviewed, almost all the presumed beneficial mutations were only beneficial in a very narrow sense- but each mutation consistently involved loss of function changes-hence loss of information. Sanford: Genetic Entropy Mutations, in summary, tend to induce sickness, death, or deficiencies. No evidence in the vast literature of heredity change shows unambiguous evidence that random mutation itself, even with geographical isolation of populations leads to speciation. Lynn Margulis - Acquiring Genomes [2003], p. 29. But there is no evidence that DNA mutations can provide the sorts of variation needed for evolution There is no evidence for beneficial mutations at the level of macroevolution, but there is also no evidence at the level of what is commonly regarded as microevolution. Jonathan Wells (PhD. - Molecular Biology) Evolution vs. Genetic Entropy - video *******www.youtube****/watch?v=mmbRbyv2PA0 Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/
27 May 2010
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