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Genetic Entropy & the Mystery of the Genome - Book Review *******www.tccsa.tc/articles/genetic_entropy.html John C. Sanford - Bio Excerpt: * Co-developer of “Mendel’s Accountant” – today’s most advanced forward-time population genetics simulation program * Primary inventor of the biolistic (gene gun) process * Co-inventor of the Pathogen-derived Resistance (PDR) process * Co-inventor of the Genetic Vaccination process * Primary inventor of numerous conventionally-bred fruit varieties * Most of the world's transgenic crop acreage were transformed via my biolistic process *******www.nysaes***rnell.edu/hort/faculty/sanford/ The entire 49 min. video by John Sanford may be ordered here: The Mystery of Our Declining Genes DVD ********store.creation****/us/product_info.php?sku=30-9-583 Natural Selection Reduces Genetic Information - Dr. Georgia Purdom - video *******www.metacafe****/watch/4036808 Natural Selection Reduces Genetic Information - No Beneficial Mutations - Spetner - Denton - video *******www.metacafe****/watch/4036816 Survival of the fittest theory: Darwinism’s limits – Jerry Fodor and Massimo Piattelli-Palmarini – Feb 2010 Excerpt: Much of the vast neo-Darwinian literature is distressingly uncritical. The possibility that anything is seriously amiss with Darwin’s account of evolution is hardly (ever) considered. ,,, Natural Selection has shown insidious imperialistic tendencies. *******www.newscientist****/article/mg20527466.100-survival-of-the-fittest-theory-darwinisms-limits.html This following study is very interesting for the researcher surveyed 130 DNA-based evolutionary trees to see if "natural selection" results in speciation and found: Accidental origins: Where species come from - March 2010 Excerpt: If speciation results from natural selection via many small changes, you would expect the branch lengths to fit a bell-shaped curve.,,, Instead, Pagel's team found that in 78 per cent of the trees, the best fit for the branch length distribution was another familiar curve, known as the exponential distribution. Like the bell curve, the exponential has a straightforward explanation - but it is a disquieting one for evolutionary biologists. The exponential is the pattern you get when you are waiting for some single, infrequent event to happen.,,,To Pagel, the implications for speciation are clear: "It isn't the accumulation of events that causes a speciation, it's single, rare events falling out of the sky, so to speak." *******www.newscientist****/article/mg20527511.400-accidental-origins-where-species-come-from.html?page=2 The Strength of Phenotypic Selection in Natural Populations This review demonstrates that our information about the strength of phenotypic selection in natural populations has increased dramatically in the past 2 decades, but many important issues about selection remain unresolved. *******www.oeb.harvard.edu/faculty/hoekstra/PDFs/Kingsolver2001AmNat.pdf EXPELLED - Natural Selection And Genetic Mutations - video *******www.metacafe****/watch/4036840 "...but Natural Selection reduces genetic information and we know this from all the Genetic Population studies that we have..." Maciej Marian Giertych - Population Geneticist - member of the European Parliament - EXPELLED Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/
7 May 2010
2063
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Genetic Entropy & the Mystery of the Genome - Book Review *******www.tccsa.tc/articles/genetic_entropy.html John C. Sanford - Bio Excerpt: * Co-developer of “Mendel’s Accountant” – today’s most advanced forward-time population genetics simulation program * Primary inventor of the biolistic (gene gun) process * Co-inventor of the Pathogen-derived Resistance (PDR) process * Co-inventor of the Genetic Vaccination process * Primary inventor of numerous conventionally-bred fruit varieties * Most of the world's transgenic crop acreage were transformed via my biolistic process *******www.nysaes***rnell.edu/hort/faculty/sanford/ This following study confirmed the "detrimental" mutation rate for humans, of 100 to 300, estimated by John Sanford in his book "Genetic Entropy" in 2005: Human mutation rate revealed: August 2009 Every time human DNA is passed from one generation to the next it accumulates 100–200 new mutations, according to a DNA-sequencing analysis of the Y chromosome. (Of note: this number is derived after "compensatory mutations") *******www.nature****/news/2009/090827/full/news.2009.864.html This mutation rate of 100 to 200 is far greater than even what evolutionists agree is an acceptable mutation rate for an organism: Beyond A 'Speed Limit' On Mutations, Species Risk Extinction Excerpt: Shakhnovich's group found that for most organisms, including viruses and bacteria, an organism's rate of genome mutation must stay below 6 mutations per genome per generation to prevent the accumulation of too many potentially lethal changes in genetic material. *******www.sciencedaily****/releases/2007/10/071001172753.htm Professional evolutionary biologists are hard-pressed to cite even one clear-cut example of evolution through a beneficial mutation to the DNA of humans which would violate the principle of genetic entropy. Although a materialist may try to claim the lactase persistence mutation as a lonely example of a "truly" beneficial mutation in humans, lactase persistence is actually a loss of a instruction in the genome to turn the lactase enzyme off, so the mutation clearly does not violate Genetic Entropy. Yet at the same time, the evidence for the detrimental nature of mutations in humans is overwhelming for doctors have already cited over 3500 mutational disorders (Dr. Gary Parker). (of note: this figure is now known to be over 6000 mendelian mutational disorders - John Sanford - 2010) "Mutations" by Dr. Gary Parker Excerpt: human beings are now subject to over 3500 mutational disorders. (Now known to be over 6000 - John Sanford - 2010) *******www.answersingenesis****/home/area/cfol/ch2-mutations.asp Using Computer Simulation to Understand Mutation Accumulation Dynamics and Genetic Load: Excerpt: We apply a biologically realistic forward-time population genetics program to study human mutation accumulation under a wide-range of circumstances. Using realistic estimates for the relevant biological parameters, we investigate the rate of mutation accumulation, the distribution of the fitness effects of the accumulating mutations, and the overall effect on mean genotypic fitness. Our numerical simulations consistently show that deleterious mutations accumulate linearly across a large portion of the relevant parameter space. *******bioinformatics.cau.edu.cn/lecture/chinaproof.pdf MENDEL’S ACCOUNTANT: J. SANFORD†, J. BAUMGARDNER‡, W. BREWER§, P. GIBSON¶, AND W. REMINE *******mendelsaccount.sourceforge**** *******www.scpe****/vols/vol08/no2/SCPE_8_2_02.pdf Human Evolution - Genetic Adam And Eve - Hugh Ross - video *******www.metacafe****/watch/4036776 Human Evolution? - The Compelling Genetic, Fossil Evidence & Tool Making For Adam and Eve - Dr. Fazale Rana - video *******www.metacafe****/watch/4284482 Does human genetic evidence support Noah's flood? Fazale Rana - video *******www.metacafe****/watch/4116168 Tracing Your Ancestors Through History - Noah's Descendants - video *******edinburghcreationgroup****/ancestors.xml TABLE OF NATIONS (GENEALOGY OF MANKIND) by Tim Osterholm Excerpt: The fact is, that wherever its statements can be sufficiently tested, Genesis 10 of the Bible has been found completely accurate; resulting partly from linguistic studies, partly from archaeology, and, more recently still, from the findings of physical anthropologists, who are, to this day, recovering important clues to lines of migration in ancient historic times. As implied in verse 32 of Genesis 10, this Table includes everybody; meaning that so-called fossil man, primitive peoples (ancient and modern) and modern man are all derived from Noah's three sons, Shem, Ham, and Japheth. *******www.soundchristian****/man/ Startling Evidence That Noah's Flood Really Happened - video *******video.google****/videoplay?docid=-7075979791519871387 Even evolutionists admit the fossil record does not reveal a gradual process of monkeys evolving into humans: Evolution of the Genus Homo - Annual Review of Earth and Planetary Sciences - Tattersall, Schwartz, May 2009 Excerpt: "Definition of the genus Homo is almost as fraught as the definition of Homo sapiens. We look at the evidence for “early Homo,” finding little morphological basis for extending our genus to any of the 2.5–1.6-myr-old fossil forms assigned to “early Homo” or Homo habilis/rudolfensis." *******arjournals.annualreviews****/doi/abs/10.1146/annurev.earth.031208.100202 "When we consider the remote past, before the origin of the actual species Homo sapiens, we are faced with a fragmentary and disconnected fossil record. Despite the excited and optimistic claims that have been made by some paleontologists, no fossil hominid species can be established as our direct ancestor." Richard Lewontin - Harvard Zoologist *******www.discovery****/a/9961 The following video is downright eye-opening with its evidence for Biblical authenticity: The Physical Ashen Remains Of Sodom and Gomorrah - video *******www.youtube****/watch?v=FwTVFk1HK3Y The entire 49 min. video by John Sanford may be ordered here: The Mystery of Our Declining Genes DVD ********store.creation****/us/product_info.php?sku=30-9-583 Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/
6 May 2010
2890
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Donald E. Johnson Bibliography *******www.amazon****/Donald-E.-Johnson/e/B001K8LWHQ Entire video: *******www.ideaclubtcw****/video/DEJohnson.html Dr. Johnson's website: Science Integrity - Exposing Unsubstantiated Science Claims *******scienceintegrity****/PNNP.aspx *******scienceintegrity****/default.aspx Cells Are Like Robust Computational Systems, - June 2009 Excerpt: "We now have reason to think of cells as robust computational devices, employing redundancy in the same way that enables large computing systems, such as Amazon, to keep operating despite the fact that servers routinely fail." *******www.sciencedaily****/releases/2009/06/090616103205.htm Nanoelectronic Transistor Combined With Biological Machine Could Lead To Better Electronics: - Aug. 2009 Excerpt: While modern communication devices rely on electric fields and currents to carry the flow of information, biological systems are much more complex. They use an arsenal of membrane receptors, channels and pumps to control signal transduction that is unmatched by even the most powerful computers. *******www.sciencedaily****/releases/2009/08/090811091834.htm Ben Stein - EXPELLED - The Complexity of the Cell - video *******www.metacafe****/watch/4018253/expelled_complexity_of_the_cell/ The Capabilities of Chaos and Complexity - David L. Abel - 2009 Excerpt: "A monstrous ravine runs through presumed objective reality. It is the great divide between physicality and formalism. On the one side of this Grand Canyon lies everything that can be explained by the chance and necessity of physicodynamics. On the other side lies those phenomena than can only be explained by formal choice contingency and decision theory—the ability to choose with intent what aspects of ontological being will be preferred, pursued, selected, rearranged, integrated, organized, preserved, and used. Physical dynamics includes spontaneous non linear phenomena, but not our formal applied-science called “non linear dynamics”(i.e. language,information). *******www.mdpi****/1422-0067/10/1/247/pdf Here, we show that the universal genetic code can efficiently carry arbitrary parallel codes much better than the vast majority of other possible genetic codes.... the present findings support the view that protein-coding regions can carry abundant parallel codes. *******genome.cshlp****/content/17/4/405.full The data compression of some stretches of human DNA is estimated to be up to 12 codes thick (Trifonov, 1989). (This is well beyond the complexity of any computer code ever written by man). John Sanford - Genetic Entropy John Sanford, a leading expert in Genetics, comments on some of the stunning poly-functional complexity found in the genome: "There is abundant evidence that most DNA sequences are poly-functional, and therefore are poly-constrained. This fact has been extensively demonstrated by Trifonov (1989). For example, most human coding sequences encode for two different RNAs, read in opposite directions i.e. Both DNA strands are transcribed ( Yelin et al., 2003). Some sequences encode for different proteins depending on where translation is initiated and where the reading frame begins (i.e. read-through proteins). Some sequences encode for different proteins based upon alternate mRNA splicing. Some sequences serve simultaneously for protein-encoding and also serve as internal transcriptional promoters. Some sequences encode for both a protein coding, and a protein-binding region. Alu elements and origins-of-replication can be found within functional promoters and within exons. Basically all DNA sequences are constrained by isochore requirements (regional GC content), “word” content (species-specific profiles of di-, tri-, and tetra-nucleotide frequencies), and nucleosome binding sites (i.e. All DNA must condense). Selective condensation is clearly implicated in gene regulation, and selective nucleosome binding is controlled by specific DNA sequence patterns - which must permeate the entire genome. Lastly, probably all sequences do what they do, even as they also affect general spacing and DNA-folding/architecture - which is clearly sequence dependent. To explain the incredible amount of information which must somehow be packed into the genome (given that extreme complexity of life), we really have to assume that there are even higher levels of organization and information encrypted within the genome. For example, there is another whole level of organization at the epigenetic level (Gibbs 2003). There also appears to be extensive sequence dependent three-dimensional organization within chromosomes and the whole nucleus (Manuelides, 1990; Gardiner, 1995; Flam, 1994). Trifonov (1989), has shown that probably all DNA sequences in the genome encrypt multiple “codes” (up to 12 codes). (Dr. John Sanford; Genetic Entropy 2005) Comprehensive Mapping of Long-Range Interactions Reveals Folding Principles of the Human Genome - Oct. - 2009 Excerpt: We identified an additional level of genome organization that is characterized by the spatial segregation of open and closed chromatin to form two genome-wide compartments. At the megabase scale, the chromatin conformation is consistent with a fractal globule, a knot-free, polymer conformation that enables maximally dense packing while preserving the ability to easily fold and unfold any genomic locus. *******www.sciencemag****/cgi/content/abstract/326/5950/289 Here is a site that gives a clear example of what Dr. Sanford means by Poly-Functional equals Poly-Contrained: Poly-Functional Complexity equals Poly-Constrained Complexity *******docs.google****/Doc?docid=0AYmaSrBPNEmGZGM4ejY3d3pfMjdoZmd2emZncQ Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/
16 Feb 2010
2217
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DNA - Poly-Functional Complexity equals Poly-Constrained Complexity *******docs.google****/Doc?docid=0AYmaSrBPNEmGZGM4ejY3d3pfMjdoZmd2emZncQ As former president of the French Academy of Sciences Pierre P. Grasse has stated: “What is the use of their unceasing mutations, if they do not change? In sum, the mutations of bacteria and viruses are merely hereditary fluctuations around a median position; a swing to the right, a swing to the left, but no final evolutionary effect.” Random Mutations Destroy Information - Perry Marshall - video *******www.metacafe****/watch/4023143 Needless to say, this limit to the variability of "simple" single cell organism, bacteria, and viruses, is extremely bad news for the materialist. Materialists simply do not have the "beneficial" mutations they need to make evolution work. The following site has numerous quotes, studies and videos which reveal the overwhelmingly negative mutation rate which has been found in life: Mutation Studies, Videos, And Quotes *******docs.google****/Doc?docid=0AYmaSrBPNEmGZGM4ejY3d3pfMjZjZnM5M21mZg Evolution vs. Genetic Entropy - video *******www.metacafe****/watch/4028086 Assessing the NCSE’s Citation Bluffs on the Evolution of New Genetic Information - Feb. 2010 *******www.evolutionnews****/2010/02/assessing_the_ncses_citation_b.html How to Play the Gene Evolution Game - Casey Luskin - Feb. 2010 *******www.evolutionnews****/2010/02/how_to_play_the_gene_evolution.html The NCSE, Judge Jones, and Bluffs About the Origin of New Functional Genetic Information - Casey Luskin - March 2010 *******www.discovery****/a/14251 It should be noted that evolutionists like to play head games with Claude Shannon's broad definition of information since "non-functional" information bits may be considered information in his broad definition, yet when looked at carefully, Shannon's work actually fully supports Intelligent Design as is illustrated in the following video and article: DNA and The Genetic Code Pt 3 - Perry Marshall - video *******www.youtube****/watch?v=FtMQUFOwEFo Skeptic's Objection to Information Theory #1: "DNA is Not a Code" *******cosmicfingerprints****/dnanotcode.htm DNA – The Genetic Code – Optimal Error Minimization & Parallel Codes – Dr. Fazale Rana – video *******www.metacafe****/watch/4491422 Here, we show that the universal genetic code can efficiently carry arbitrary parallel codes much better than the vast majority of other possible genetic codes…. the present findings support the view that protein-coding regions can carry abundant parallel codes. *******genome.cshlp****/content/17/4/405.full The data compression of some stretches of human DNA is estimated to be up to 12 codes thick (12 different ways of DNA transcription) (Trifonov, 1989). (This is well beyond the complexity of any computer code ever written by man). John Sanford – Genetic Entropy The multiple codes of nucleotide sequences. Trifonov EN. – 1989 Excerpt: Nucleotide sequences carry genetic information of many different kinds, not just instructions for protein synthesis (triplet code). *******www.ncbi.nlm.nih.gov/pubmed/2673451 Dr. Jerry Bergman, “Divine Engineering: Unraveling DNA’s Design”: The DNA packing process is both complex and elegant and is so efficient that it achieves a reduction in length of DNA by a factor of 1 million. DNA Wrapping (Histone Protein Wrapping to Cell Division)- video *******www.youtube****/watch?v=gbSIBhFwQ4s Human DNA is like a computer program but far, far more advanced than any software we’ve ever created. Bill Gates, The Road Ahead, 1996, p. 188 The Coding Found In DNA Surpasses Man’s Ability To Code – Stephen Meyer – video *******www.metacafe****/watch/4050638 Bill Gates, in recognizing the superiority found in Genetic Coding, compared to the best computer coding we now have, has now funded research into this area: Welcome to CoSBi – (Computational and Systems Biology) Excerpt: Biological systems are the most parallel systems ever studied and we hope to use our better understanding of how living systems handle information to design new computational paradigms, programming languages and software development environments. The net result would be the design and implementation of better applications firmly grounded on new computational, massively parallel paradigms in many different areas. Human Genome “Infinitely More Complex” Than Expected – April 2010 Excerpt: Hayden acknowledged that the “junk DNA” paradigm has been blown to smithereens. “Just one decade of post-genome biology has exploded that view,” she said, speaking of the gene regulation was a straightforward, linear process of gene coding for regulator protein that controls transcription. “Biology’s new glimpse at a universe of non-coding DNA – what used to be called ‘junk’ DNA – has been fascinating and befuddling.” If it’s junk, why would the human body decode 74% to 93& of it? Inside the Human Genome: A Case for Non-Intelligent Design – Pg. 57 By John C. Avise Excerpt: “Another compilation of gene lesions responsible for inherited diseases is the web-based Human Gene Mutation Database (HGMD). Recent versions of HGMD describe more than 75,000 different disease causing mutations identified to date in Homo-sapiens.” HGMD®: Now celebrating our 100,000 mutation milestone! *******www.hgmd.cf.ac***/ac/index.php I really question their use of the word “celebrating”. The burning question is where in the world are all the hypothetical beneficial mutations??? The two most popular examples given Sickle cell and Lactase persistence, Sickle Cell is only beneficial in a limited sense and clearly detrimental in a molecular sense. Whereas Lactase persistence is the same in that it confers an advantage but it loses information in the genome for turning the lactase enzyme off. thus both of the most popular examples cited by evolutionists lose functional information. Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/
14 May 2010
1946
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The foundational rule for biology, Genetic Entropy, which can draw its foundation in science from the twin pillars of the Second Law of Thermodynamics and from the Law of Conservation of Information (Dembski, Marks) (Abel - Null Hypothesis), can be stated something like this: "All beneficial adaptations away from a parent species for a sub-species, which increase fitness to a particular environment, will always come at a loss of the optimal functional information that was originally created in the parent species genome." _ Genetic Entropy is the true rule for all biological adaptations - The malaria parasite, due to its comparatively enormous population size, has in 1 year more mutation/duplication/selection events than all mammal lineages have had in the entire +100 million years they have been in the fossil record. Moreover, since single cell organisms and viruses replicate, and mutate/duplicate, far more quickly than multi-cellular life-forms can, scientists can do experiments on single celled organisms and viruses to see what we can actually expect to happen over millions of years for mammals with far smaller population sizes. Malaria and AIDS are among the largest real world tests that can be performed to see if evolutionary presumptions are true. "Indeed, the work on malaria and AIDS demonstrates that after all possible unintelligent processes in the cell--both ones we've discovered so far and ones we haven't--at best extremely limited benefit, since no such process was able to do much of anything. It's critical to notice that no artificial limitations were placed on the kinds of mutations or processes the microorganisms could undergo in nature. Nothing--neither point mutation, deletion, insertion, gene duplication, transposition, genome duplication, self-organization nor any other process yet undiscovered--was of much use." Michael Behe, The Edge of Evolution, pg. 162 Swine Flu, Viruses, and the Edge of Evolution *******www.evolutionnews****/2009/05/swine_flu_viruses_and_the_edge.html A review of The Edge of Evolution: The Search for the Limits of Darwinism by Michael J. Behe The numbers of Plasmodium and HIV in the last 50 years greatly exceeds the total number of mammals since their supposed evolutionary origin (several hundred million years ago), yet little has been achieved by evolution. This suggests that mammals could have "invented" little in their time frame. Behe: Our experience with HIV gives good reason to think that Darwinism doesnt do much—even with billions of years and all the cells in that world at its disposal (p. 155). *******creation****/review-michael-behe-edge-of-evolution Behe and Snoke go even further in addressing the Gene Duplication scenario in this following study: Simulating evolution by gene duplication of protein features that require multiple amino acid residues: Michael J. Behe and David W. Snoke Excerpt: Gene duplication is thought to be a major source of evolutionary innovation because it allows one copy of a gene to mutate and explore genetic space while the other copy continues to fulfill the original function. (However), At smaller population sizes, the time to fixation varies linearly with 1/N and exceeds the inverse of the point mutation rate. We conclude that, in general, to be fixed in 10^8 generations, the production of novel protein features that require the participation of two or more amino acid residues simply by multiple point mutations in duplicated genes would entail population sizes of no less than 10^9. *******www.pubmedcentral.nih.gov/articlerender.fcgi?artid=2286568 Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/
28 Sep 2010
2684
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"I have seen estimates of the incidence of the ratio of deleterious-to-beneficial mutations which range from one in one thousand up to one in one million. The best estimates seem to be one in one million (Gerrish and Lenski, 1998). The actual rate of beneficial mutations is so extremely low as to thwart any actual measurement (Bataillon, 2000, Elena et al, 1998). Therefore, I cannot ...accurately represent how rare such beneficial mutations really are." (J.C. Sanford; Genetic Entropy page 24) - 2005 Estimation of spontaneous genome-wide mutation rate parameters: whither beneficial mutations? (Thomas Bataillon) Abstract......It is argued that, although most if not all mutations detected in mutation accumulation experiments are deleterious, the question of the rate of favourable mutations (and their effects) is still a matter for debate. *******www.nature****/hdy/journal/v84/n5/full/6887270a.html Distribution of fitness effects caused by random insertion mutations in Escherichia coli Excerpt: At least 80% of the mutations had a significant negative effect on fitness, whereas none of the mutations had a significant positive effect. *******www.springerlink****/content/r37w1hrq5l0q3832/ High Frequency of Cryptic Deleterious Mutations in Caenorhabditis elegans ( Esther K. Davies, Andrew D. Peters, Peter D. Keightley) "In fitness assays, only about 4 percent of the deleterious mutations fixed in each line were detectable. The remaining 96 percent, though cryptic, are significant for mutation load...the presence of a large class of mildly deleterious mutations can never be ruled out. " *******www.sciencemag****/cgi/content/abstract/285/5434/1748 But in all the reading Ive done in the life-sciences literature, Ive never found a mutation that added information All point mutations that have been studied on the molecular level turn out to reduce the genetic information and not increase it. Lee Spetner - Ph.D. Physics - MIT - (Not By Chance: Shattering the Modern Theory of Evolution) "Bergman (2004) has studied the topic of beneficial mutations. Among other things, he did a simple literature search via Biological Abstracts and Medline. He found 453,732 mutation hits, but among these only 186 mentioned the word beneficial (about 4 in 10,000). When those 186 references were reviewed, almost all the presumed beneficial mutations were only beneficial in a very narrow sense- but each mutation consistently involved loss of function changes-hence loss of information. Sanford: Genetic Entropy Mutations, in summary, tend to induce sickness, death, or deficiencies. No evidence in the vast literature of heredity change shows unambiguous evidence that random mutation itself, even with geographical isolation of populations leads to speciation. Lynn Margulis - Acquiring Genomes [2003], p. 29. But there is no evidence that DNA mutations can provide the sorts of variation needed for evolution There is no evidence for beneficial mutations at the level of macroevolution, but there is also no evidence at the level of what is commonly regarded as microevolution. Jonathan Wells (PhD. - Molecular Biology) Evolution vs. Genetic Entropy - video *******www.youtube****/watch?v=mmbRbyv2PA0 Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/
27 May 2010
2624
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8:25
‘Darwinian evolution is impossible’ - John Sanford - Geneticist ‘Mutations are word-processing errors in the cell’s instruction manual. Mutations systematically destroy genetic information—even as word processing errors destroy written information. While there are some rare beneficial mutations (Even the rare beneficial mutations still lose information), bad mutations outnumber them—perhaps by a million to one. So even allowing for beneficial mutations, the net effect of mutation is overwhelmingly deleterious. The more the mutations, the less the information. This is fundamental to the mutation process.’ *******creation****/geneticist-evolution-impossible Random Mutations Destroy Information - Perry Marshall - video *******www.metacafe****/watch/4023143/random_mutations_destroy_information_perry_marshall/ "The neo-Darwinians would like us to believe that large evolutionary changes can result from a series of small events if there are enough of them. But if these events all lose information they can’t be the steps in the kind of evolution the neo-Darwin theory is supposed to explain, no matter how many mutations there are. Whoever thinks macroevolution can be made by mutations that lose information is like the merchant who lost a little money on every sale but thought he could make it up on volume." Lee Spetner (Ph.D. Physics - MIT - Not By Chance) “But there is no evidence that DNA mutations can provide the sorts of variation needed for evolution… There is no evidence for beneficial mutations at the level of macroevolution, but there is also no evidence at the level of what is commonly regarded as microevolution.” Jonathan Wells (PhD. - Molecular Biology) As former president of the French Academy of Sciences Pierre P. Grasse has stated: “What is the use of their unceasing mutations, if they do not change? In sum, the mutations of bacteria and viruses are merely hereditary fluctuations around a median position; a swing to the right, a swing to the left, but no final evolutionary effect.” The Sheer Lack Of Evidence For Macro Evolution - William Lane Craig - video *******www.metacafe****/watch/4023134/the_sheer_lack_of_evidence_for_macro_evolution_william_lane_craig/ In fact, from consistent findings such as these, it is increasingly apparent the principle of Genetic Entropy is the overriding foundational rule for all of biology, with no exceptions at all, and belief in "truly" beneficial mutations is nothing more than wishful speculation on the materialist part which has no foundation in empirical science whatsoever. The Capabilities of Chaos and Complexity: David L. Abel - Null Hypothesis For Information Generation - 2009 To focus the scientific community’s attention on its own tendencies toward overzealous metaphysical imagination bordering on “wish-fulfillment,” we propose the following readily falsifiable null hypothesis, and invite rigorous experimental attempts to falsify it: "Physicodynamics cannot spontaneously traverse The Cybernetic Cut: physicodynamics alone cannot organize itself into formally functional systems requiring algorithmic optimization, computational halting, and circuit integration." A single exception of non trivial, unaided spontaneous optimization of formal function by truly natural process would falsify this null hypothesis. *******www.mdpi****/1422-0067/10/1/247/pdf *******mdpi****/1422-0067/10/1/247/ag Evolution vs. Genetic Entropy - video *******www.metacafe****/watch/4028086/evolution_vs_genetic_entropy/ Here is a site that gives a clear example of what Dr. Sanford means by Poly-Functional equals Poly-Contrained: Poly-Functional Complexity equals Poly-Constrained Complexity *******docs.google****/Doc?docid=0AYmaSrBPNEmGZGM4ejY3d3pfMjdoZmd2emZncQ For a broad outline of the "Fitness test", required to be passed to show a violation of the principle of Genetic Entropy, please see the following video and articles: Is Antibiotic Resistance evidence for evolution? - "The Fitness Test" - video *******www.metacafe****/watch/3995248/is_antibiotic_resistance_evidence_for_evolution_the_fitness_test/ Testing the Biological Fitness of Antibiotic Resistant Bacteria - 2008 *******www.answersingenesis****/articles/aid/v2/n1/darwin-at-drugstore List Of Degraded Molecular Abilities Of Antibiotic Resistant Bacteria: *******www.trueorigin****/bacteria01.asp The foundational rule of Genetic Entropy for biology, which can draw its foundation in science from the twin pillars of the Second Law of Thermodynamics and from the Law of Conservation of Information (Dembski, Marks), can be stated something like this: "All beneficial adaptations away from a parent species for a sub-species, which increase fitness to a particular environment, will always come at a loss of the optimal functional information that was originally created in the parent species genome." Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/ So where did the information come from if not by mutations? John 1:1 "In the beginning was The Word,,,
3 Oct 2011
3022
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5:10
The integrated complexity Flew alludes to in his conversion is no small matter. The gulf between the integrated complexity of living organisms and of the most advanced machines man has made is enormous. The Cell as a Collection of Protein Machines "We have always underestimated cells. Undoubtedly we still do today,,, Indeed, the entire cell can be viewed as a factory that contains an elaborate network of interlocking assembly lines, each which is composed of a set of large protein machines." Bruce Alberts: Former President, National Academy of Sciences; *******www.imbb.forth.gr/people/aeconomou/documents/Alberts98.pdf Map Of Major Metabolic Pathways In A Cell - Diagram ********webspace.utexas.edu/yg387/MetabolicPathway.jpg The Capabilities of Chaos and Complexity - David L. Abel - 2009 Excerpt: "A monstrous ravine runs through presumed objective reality. It is the great divide between physicality and formalism. On the one side of this Grand Canyon lies everything that can be explained by the chance and necessity of physicodynamics. On the other side lies those phenomena than can only be explained by formal choice contingency and decision theory—the ability to choose with intent what aspects of ontological being will be preferred, pursued, selected, rearranged, integrated, organized, preserved, and used. Physical dynamics includes spontaneous non linear phenomena, but not our formal applied-science called “non linear dynamics”(i.e. language,information). *******www.mdpi****/1422-0067/10/1/247/pdf 3-D Structure Of Human Genome: Fractal Globule Architecture Packs Two Meters Of DNA Into Each Cell - Oct. 2009 Excerpt: the information density in the nucleus is trillions of times higher than on a computer chip -- while avoiding the knots and tangles that might interfere with the cell's ability to read its own genome. Moreover, the DNA can easily unfold and refold during gene activation, gene repression, and cell replication. *******www.sciencedaily****/releases/2009/10/091008142957.htm The data compression of some stretches of human DNA is estimated to be up to 12 codes thick (Trifonov, 1989). (This is well beyond the complexity of any computer code ever written by man). John Sanford - Genetic Entropy "There is abundant evidence that most DNA sequences are poly-functional, and therefore are poly-constrained. This fact has been extensively demonstrated by Trifonov (1989). For example, most human coding sequences encode for two different RNAs, read in opposite directions i.e. Both DNA strands are transcribed ( Yelin et al., 2003). Some sequences encode for different proteins depending on where translation is initiated and where the reading frame begins (i.e. read-through proteins). Some sequences encode for different proteins based upon alternate mRNA splicing. Some sequences serve simultaneously for protein-encoding and also serve as internal transcriptional promoters. Some sequences encode for both a protein coding, and a protein-binding region. Alu elements and origins-of-replication can be found within functional promoters and within exons. Basically all DNA sequences are constrained by isochore requirements (regional GC content), “word” content (species-specific profiles of di-, tri-, and tetra-nucleotide frequencies), and nucleosome binding sites (i.e. All DNA must condense). Selective condensation is clearly implicated in gene regulation, and selective nucleosome binding is controlled by specific DNA sequence patterns - which must permeate the entire genome. Lastly, probably all sequences do what they do, even as they also affect general spacing and DNA-folding/architecture - which is clearly sequence dependent. To explain the incredible amount of information which must somehow be packed into the genome (given that extreme complexity of life), we really have to assume that there are even higher levels of organization and information encrypted within the genome. For example, there is another whole level of organization at the epigenetic level (Gibbs 2003). There also appears to be extensive sequence dependent three-dimensional organization within chromosomes and the whole nucleus (Manuelides, 1990; Gardiner, 1995; Flam, 1994). Trifonov (1989), has shown that probably all DNA sequences in the genome encrypt multiple “codes” (up to 12 codes). (Dr. John Sanford; Genetic Entropy 2005) Here is a site that gives a clear example of what Dr. Sanford means by Poly-Functional equals Poly-Contrained: Poly-Functional Complexity equals Poly-Constrained Complexity *******docs.google****/Doc?docid=0AYmaSrBPNEmGZGM4ejY3d3pfMjdoZmd2emZncQ Journey Inside The Cell - DNA to mRNA to Proteins - Stephen Meyer - Signature In The Cell - video *******www.youtube****/watch?v=1fiJupfbSpg The Capabilities of Chaos and Complexity: David L. Abel - Null Hypothesis For Information Generation - 2009 To focus the scientific community’s attention on its own tendencies toward overzealous metaphysical imagination bordering on “wish-fulfillment,” we propose the following readily falsifiable null hypothesis, and invite rigorous experimental attempts to falsify it: "Physicodynamics cannot spontaneously traverse The Cybernetic Cut: physicodynamics alone cannot organize itself into formally functional systems requiring algorithmic optimization, computational halting, and circuit integration." A single exception of non trivial, unaided spontaneous optimization of formal function by truly natural process would falsify this null hypothesis. *******www.mdpi****/1422-0067/10/1/247/pdf *******mdpi****/1422-0067/10/1/247/ag Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/
11 Jan 2011
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Stephen C. Meyer - Signature In The Cell: "DNA functions like a software program," "We know from experience that software comes from programmers. Information--whether inscribed in hieroglyphics, written in a book or encoded in a radio signal--always arises from an intelligent source. So the discovery of digital code in DNA provides evidence that the information in DNA also had an intelligent source." *******www.evolutionnews****/2009/07/leading_advocate_of_intelligen.html Skeptic's Objection to Information Theory #1: "DNA is Not a Code" *******cosmicfingerprints****/dnanotcode.htm Biophysicist Hubert Yockey determined that natural selection would have to explore 1.40 x 10^70 different genetic codes to discover the optimal universal genetic code that is found in nature. The maximum amount of time available for it to originate is 6.3 x 10^15 seconds. Natural selection would have to evaluate roughly 10^55 codes per second to find the one that is optimal. Put simply, natural selection lacks the time necessary to find the optimal universal genetic code we find in nature. (Fazale Rana, -The Cell’s Design – 2008 – page 177) DNA: The Alphabet of Life - David Klinghoffer Excerpt: But all this is trivial compared to the largely unheralded insight gained from the Human Genome Project, completed in 2003. The insight is disturbing. It is that while DNA codes for the cell's building blocks, the information needed to build the rest of the creature is seemingly, in large measure, absent. ,,,The physically encoded information to form that mouse, as opposed to that fly, isn't there. Instead, "It is as if the 'idea' of the fly (or any other organism) must somehow permeate the genome that gives rise to it." *******www.evolutionnews****/2009/07/dna_the_alphabet_of_life.html Stephen Meyer is interviewed about the "information problem" in DNA, Signature in the Cell - video *******downloads.cbn****/cbnnewsplayer/cbnplayer.swf?aid=8497 The DNA Enigma - Where Did The Information Come Frome? - Stephen C. Meyer - video *******www.metacafe****/watch/4125886/the_dna_enigma_where_did_the_information_come_frome_stephen_c_meyer/ Stephen Meyer - Functional Proteins And Information For Body Plans - video *******www.metacafe****/watch/4050681/stephen_meyer_functional_proteins_and_information_for_body_plans/ Fearfully and Wonderfully Made - Glimpses At Human Development In The Womb - video *******www.metacafe****/watch/4249713/fearfully_and_wonderfully_made_glimpses_at_development_in_the_womb/ Jeremiah 1:5 Before I formed you in the womb I knew you, before you were born I set you apart;,, The Origin of Biological Information and the Higher Taxonomic Categories - Stephen Meyer Excerpt: "Neo-Darwinism seeks to explain the origin of new information, form, and structure as a result of selection acting on randomly arising variation at a very low level within the biological hierarchy, mainly, within the genetic text. Yet the major morphological innovations depend on a specificity of arrangement at a much higher level of the organizational hierarchy, a level that DNA alone does not determine. Yet if DNA is not wholly responsible for body plan morphogenesis, then DNA sequences can mutate indefinitely, without regard to realistic probabilistic limits, and still not produce a new body plan. Thus, the mechanism of natural selection acting on random mutations in DNA cannot in principle generate novel body plans, including those that first arose in the Cambrian explosion." *******eyedesignbook****/ch6/eyech6-append-d.html In fact the coding found in DNA far surpasses any code man has ever devised: Higher Levels Of Information In Life - Stephen Meyer - video *******www.metacafe****/watch/4050638/the_coding_found_in_dna_surpasses_mans_ability_to_code_stephen_meyer/ The human genome, according to Bill Gates the founder of Microsoft, far, far surpasses, in complexity, any computer program ever written by man. The data compression (multiple meanings) of some stretches of human DNA is estimated to be up to 12 codes thick! (Trifonov, 1989) No line of computer code ever written by man approaches that level of data compression (poly-functional complexity). Here, we show that the universal genetic code can efficiently carry arbitrary parallel codes much better than the vast majority of other possible genetic codes.... the present findings support the view that protein-coding regions can carry abundant parallel codes. *******genome.cshlp****/content/17/4/405.full John Sanford, a leading expert in Genetics, comments on some of the stunning poly-functional complexity found in the genome: "There is abundant evidence that most DNA sequences are poly-functional, and therefore are poly-constrained. This fact has been extensively demonstrated by Trifonov (1989). For example, most human coding sequences encode for two different RNAs, read in opposite directions i.e. Both DNA strands are transcribed ( Yelin et al., 2003). Some sequences encode for different proteins depending on where translation is initiated and where the reading frame begins (i.e. read-through proteins). Some sequences encode for different proteins based upon alternate mRNA splicing. Some sequences serve simultaneously for protein-encoding and also serve as internal transcriptional promoters. Some sequences encode for both a protein coding, and a protein-binding region. Alu elements and origins-of-replication can be found within functional promoters and within exons. Basically all DNA sequences are constrained by isochore requirements (regional GC content), “word” content (species-specific profiles of di-, tri-, and tetra-nucleotide frequencies), and nucleosome binding sites (i.e. All DNA must condense). Selective condensation is clearly implicated in gene regulation, and selective nucleosome binding is controlled by specific DNA sequence patterns - which must permeate the entire genome. Lastly, probably all sequences do what they do, even as they also affect general spacing and DNA-folding/architecture - which is clearly sequence dependent. To explain the incredible amount of information which must somehow be packed into the genome (given that extreme complexity of life), we really have to assume that there are even higher levels of organization and information encrypted within the genome. For example, there is another whole level of organization at the epigenetic level (Gibbs 2003). There also appears to be extensive sequence dependent three-dimensional organization within chromosomes and the whole nucleus (Manuelides, 1990; Gardiner, 1995; Flam, 1994). Trifonov (1989), has shown that probably all DNA sequences in the genome encrypt multiple “codes” (up to 12 codes). (Dr. John Sanford; Genetic Entropy 2005) Comprehensive Mapping of Long-Range Interactions Reveals Folding Principles of the Human Genome - Oct. - 2009 Excerpt: We identified an additional level of genome organization that is characterized by the spatial segregation of open and closed chromatin to form two genome-wide compartments. At the megabase scale, the chromatin conformation is consistent with a fractal globule, a knot-free, polymer conformation that enables maximally dense packing while preserving the ability to easily fold and unfold any genomic locus. *******www.sciencemag****/cgi/content/abstract/326/5950/289 Here is a site that gives a clear example of what Dr. Sanford means by Poly-Functional equals Poly-Contrained: Poly-Functional Complexity equals Poly-Constrained Complexity *******docs.google****/Doc?docid=0AYmaSrBPNEmGZGM4ejY3d3pfMjdoZmd2emZncQ 3-D Structure Of Human Genome: Fractal Globule Architecture Packs Two Meters Of DNA Into Each Cell - Oct. 2009 Excerpt: the information density in the nucleus is trillions of times higher than on a computer chip -- while avoiding the knots and tangles that might interfere with the cell's ability to read its own genome. Moreover, the DNA can easily unfold and refold during gene activation, gene repression, and cell replication. *******www.sciencedaily****/releases/2009/10/091008142957.htm Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/
19 Jul 2010
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Biophysicist Hubert Yockey determined that natural selection would have to explore 1.40 x 10^70 different genetic codes to discover the optimal universal genetic code that is found in nature. The maximum amount of time available for it to originate is 6.3 x 10^15 seconds. Natural selection would have to evaluate roughly 10^55 codes per second to find the one that is optimal. Put simply, natural selection lacks the time necessary to find the optimal universal genetic code we find in nature. (Fazale Rana, -The Cell's Design - 2008 - page 177) Ode to the Code - Brian Hayes The few variant codes known in protozoa and organelles are thought to be offshoots of the standard code, but there is no evidence that the changes to the codon table offer any adaptive advantage. In fact, Freeland, Knight, Landweber and Hurst found that the variants are inferior or at best equal to the standard code. It seems hard to account for these facts without retreating at least part of the way back to the frozen-accident theory, conceding that the code was subject to change only in a former age of miracles, which we'll never see again in the modern world. ********www.americanscientist****/issues/pub/ode-to-the-code/4 Deciphering Design in the Genetic Code Excerpt: When researchers calculated the error-minimization capacity of one million randomly generated genetic codes, they discovered that the error-minimization values formed a distribution where the naturally occurring genetic code's capacity occurred outside the distribution. Researchers estimate the existence of 10 possible genetic codes possessing the same type and degree of redundancy as the universal genetic code. All of these codes fall within the error-minimization distribution. This finding means that of the 10 possible genetic codes, few, if any, have an error-minimization capacity that approaches the code found universally in nature. *******www.reasons****/biology/biochemical-design/fyi-id-dna-deciphering-design-genetic-code Collective evolution and the genetic code - 2006: Excerpt: The genetic code could well be optimized to a greater extent than anything else in biology and yet is generally regarded as the biological element least capable of evolving. *******www.pnas****/content/103/28/10696.full The coding system used for living beings is optimal from an engineering standpoint. Werner Gitt, - In The Beginning Was Information - p. 95 Here, we show that the universal genetic code can efficiently carry arbitrary parallel codes much better than the vast majority of other possible genetic codes.... the present findings support the view that protein-coding regions can carry abundant parallel codes. *******genome.cshlp****/content/17/4/405.full The data compression of some stretches of human DNA is estimated to be up to 12 codes thick (Trifonov, 1989). (This is well beyond the complexity of any computer code ever written by man). John Sanford - Genetic Entropy As well there are additional levels of more nuanced codes associated with manipulating DNA: Histone Inspectors: Codes and More Codes - Cornelius Hunter - March 2010 Excerpt: By now most people know about the DNA code. A DNA strand consists of a sequence of molecules, or letters, that encodes for proteins. Many people do not realize, however, that there are additional, more nuanced, codes associated with the DNA. For instance, minor chemical modifications (such as the addition of a methyl group) to the DNA provide bar-code like signals to the protein machinery that operate on the DNA. This DNA methylation influences which genes, along the DNA strand, are read off. And this DNA methylation itself may be modified to provide additional information. Or again, the DNA is wrapped around histone proteins, and these histones are also bar-coded. The histones have a hub, around which the DNA wraps, and a tail that sticks out on which chemical tags are attached. Again these tags are signals for the protein machinery. Furthermore, these tags are removed as well. Such modifications and removal of these chemical tags means that these codes are dynamic, and there are protein inspectors that double-check these complex encodings. These subtle codes are also context dependent. In one type of cell a histone modification may turn off a gene whereas in another type of cell the same histone modification may turn on the gene. *******darwins-god.blogspot****/2010/03/histone-inspectors-codes-and-more-codes.html Histone Variants: The Incredible Story of Gene Regulation *******www.uncommondescent****/intelligent-design/histone-variants-the-incredible-story-of-gene-regulation/ Human DNA is like a computer program but far, far more advanced than any software we've ever created. Bill Gates, The Road Ahead, 1996, p. 188 The Coding Found In DNA Surpasses Man's Ability To Code - Stephen Meyer - video *******www.metacafe****/watch/4050638 Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/
19 Apr 2010
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Entire video: *******www.mefeedia****/watch/32748037 Bacterial Flagellum - A Sheer Wonder Of Intelligent Design - video *******www.metacafe****/watch/3994630 Michael Behe on Falsifying Intelligent Design - video *******www.youtube****/watch?v=N8jXXJN4o_A Astonishingly, actual motors, which far surpass man-made motors in 'engineering parameters', are now being found inside 'simple cells'. Articles and Videos on Molecular Motors *******docs.google****/Doc?docid=0AYmaSrBPNEmGZGM4ejY3d3pfMzlkNjYydmRkZw&hl=en "There are no detailed Darwinian accounts for the evolution of any fundamental biochemical or cellular system only a variety of wishful speculations. It is remarkable that Darwinism is accepted as a satisfactory explanation of such a vast subject." James Shapiro - Molecular Biologist “The response I have received from repeating Behe's claim about the evolutionary literature, which simply brings out the point being made implicitly by many others, such as Chris Dutton and so on, is that I obviously have not read the right books. There are, I am sure, evolutionists who have described how the transitions in question could have occurred.” And he continues, “When I ask in which books I can find these discussions, however, I either get no answer or else some titles that, upon examination, do not, in fact, contain the promised accounts. That such accounts exist seems to be something that is widely known, but I have yet to encounter anyone who knows where they exist.” David Ray Griffin - retired professor of philosophy of religion and theology Michael Behe - Life Reeks Of Design - 2010 - video *******www.metacafe****/watch/5066181 William Dembski PhD., and Robert Marks PhD., website with peer reviewed 'Conservation Of Information' Papers (caution heavy math): *******www.evoinfo****/ Dr. Behe states in The Edge of Evolution on page 135: "Generating a single new cellular protein-protein binding site (in other words, generating a truly beneficial mutational event that would actually explain the generation of the complex molecular machinery we see in life) is of the same order of difficulty or worse than the development of chloroquine resistance in the malarial parasite." That order of difficulty is put at 10^20 replications of the malarial parasite by Dr. Behe. This number comes from direct empirical observation. Richard Dawkins’ The Greatest Show on Earth Shies Away from Intelligent Design but Unwittingly Vindicates Michael Behe - Oct. 2009 Excerpt: The rarity of chloroquine resistance is not in question. In fact, Behe’s statistic that it occurs only once in every 10^20 cases was derived from public health statistical data, published by an authority in the Journal of Clinical Investigation. The extreme rareness of chloroquine resistance is not a negotiable data point; it is an observed fact. *******www.evolutionnews****/2009/10/richard_dawkins_the_greatest_s.html Waiting Longer for Two Mutations - Michael J. Behe Excerpt: Citing malaria literature sources (White 2004) I had noted that the de novo appearance of chloroquine resistance in Plasmodium falciparum was an event of probability of 1 in 10^20. I then wrote that ‘‘for humans to achieve a mutation like this by chance, we would have to wait 100 million times 10 million years’’ (Behe 2007) (because that is the extrapolated time that it would take to produce 10^20 humans). Durrett and Schmidt (2008, p. 1507) retort that my number ‘‘is 5 million times larger than the calculation we have just given’’ using their model (which nonetheless "using their model" gives a prohibitively long waiting time of 216 million years). Their criticism compares apples to oranges. My figure of 10^20 is an empirical statistic from the literature; it is not, as their calculation is, a theoretical estimate from a population genetics model. *******www.discovery****/a/9461 This following calculation by geneticist John Sanford for 'fixing' a beneficial mutation, or for creating a new gene, in humans, gives equally absurd numbers that once again render the Darwinian scenario of humans evolving from apes completely false: Dr. Sanford calculates it would take 12 million years to “fix” a single base pair mutation into a population. He further calculates that to create a gene with 1000 base pairs, it would take 12 million x 1000 or 12 billion years. This is obviously too slow to support the creation of the human genome containing 3 billion base pairs. *******www.detectingtruth****/?p=66 Whale Evolution Vs. Population Genetics - Richard Sternberg PhD. in Evolutionary Biology - video *******www.metacafe****/watch/4165203 An Atheist Interviews Michael Behe About "The Edge Of Evolution" - video *******www.in****/videos/watchvideo-bloggingheads-interview-with-michael-behe-4734623.html Michael Behe's Amazon Blog *******www.amazon****/gp/blog/A3DGRQ0IO7KYQ2/ref=cm_blog_blog/180-3515567-6480413 Michael Behe - bio. and list of peer reviewed papers: *******www.lehigh.edu/~inbios/faculty/behe.html Should Intelligent Design Be Taught as Science? Michael Behe debates Stephen Barr - 2010 - video *******www.isi****/lectures/flvplayer/lectureplayer.aspx?file=v000355_cicero_040710.mp4&dir=mp4/lectures Main page - with audio of debate *******www.isi****/lectures/lectures.aspx?SBy=lecture&SFor=18fdfd28-e682-421f-9acf-2940402af8e3 Peer-Reviewed & Peer-Edited Scientific Publications Supporting the Theory of Intelligent Design (Annotated) - updated regularly *******www.discovery****/a/2640 Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/2009/10/intelligent-design-anthropic-hypothesis_19.html Stephen Meyer - Functional Proteins And Information For Body Plans - video *******www.metacafe****/watch/4050681 This following video is a bit more clear for explaining exactly why mutations to the DNA do not control Body Plan morphogenesis, since the mutations are the ‘bottom rung of the ladder’ as far as the 'higher levels of the layered information’ of the cell are concerned: Stephen Meyer on Craig Venter, Complexity Of The Cell & Layered Information *******www.metacafe****/watch/4798685
7 Oct 2010
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Stephen C. Meyer - The Scientific Basis For Intelligent Design - video *******www.metacafe****/watch/4104651/ Stephen C. Meyer - Signature In The Cell: "DNA functions like a software program," "We know from experience that software comes from programmers. Information--whether inscribed in hieroglyphics, written in a book or encoded in a radio signal--always arises from an intelligent source. So the discovery of digital code in DNA provides evidence that the information in DNA also had an intelligent source." *******www.evolutionnews****/2009/07/leading_advocate_of_intelligen.html Michael Behe on Falsifying Intelligent Design - video *******www.youtube****/watch?v=N8jXXJN4o_A ,,,we must concede that there are presently no detailed Darwinian accounts of the evolution of any biochemical or cellular system, only a variety of wishful speculations.’ Franklin M. Harold,* 2001. The way of the cell: molecules, organisms and the order of life, Oxford University Press, New York, p. 205. *Professor Emeritus of Biochemistry, Colorado State University, USA Michael Behe - No Scientific Literature For Evolution of Any Irreducibly Complex Molecular Machines *******www.metacafe****/watch/5302950/ Astonishingly, actual motors, which far surpass man-made motors in 'engineering parameters', are now being found inside 'simple cells'. Bacterial Flagellum - A Sheer Wonder Of Intelligent Design - video *******www.metacafe****/watch/3994630 Bacterial Flagella: A Paradigm for Design – Scott Minnich – Video *******www.vimeo****/9032112 Articles and Videos on Molecular Motors *******docs.google****/Doc?docid=0AYmaSrBPNEmGZGM4ejY3d3pfMzlkNjYydmRkZw&hl=en There are no detailed Darwinian accounts for the evolution of any fundamental biochemical or cellular system only a variety of wishful speculations. It is remarkable that Darwinism is accepted as a satisfactory explanation of such a vast subject." James Shapiro - Molecular Biologist “The response I have received from repeating Behe's claim about the evolutionary literature, which simply brings out the point being made implicitly by many others, such as Chris Dutton and so on, is that I obviously have not read the right books. There are, I am sure, evolutionists who have described how the transitions in question could have occurred.” And he continues, “When I ask in which books I can find these discussions, however, I either get no answer or else some titles that, upon examination, do not, in fact, contain the promised accounts. That such accounts exist seems to be something that is widely known, but I have yet to encounter anyone who knows where they exist.” David Ray Griffin - retired professor of philosophy of religion and theology Michael Behe - Life Reeks Of Design - 2010 - video *******www.metacafe****/watch/5066181 William Dembski PhD., and Robert Marks PhD., website with peer reviewed 'Conservation Of Information' Papers (caution heavy math): *******www.evoinfo****/ Dr. Behe states in The Edge of Evolution on page 135: "Generating a single new cellular protein-protein binding site (in other words, generating a truly beneficial mutational event that would actually explain the generation of the complex molecular machinery we see in life) is of the same order of difficulty or worse than the development of chloroquine resistance in the malarial parasite." That order of difficulty is put at 10^20 replications of the malarial parasite by Dr. Behe. This number comes from direct empirical observation. Richard Dawkins’ The Greatest Show on Earth Shies Away from Intelligent Design but Unwittingly Vindicates Michael Behe - Oct. 2009 Excerpt: The rarity of chloroquine resistance is not in question. In fact, Behe’s statistic that it occurs only once in every 10^20 cases was derived from public health statistical data, published by an authority in the Journal of Clinical Investigation. The extreme rareness of chloroquine resistance is not a negotiable data point; it is an observed fact. *******www.evolutionnews****/2009/10/richard_dawkins_the_greatest_s.html Waiting Longer for Two Mutations - Michael J. Behe Excerpt: Citing malaria literature sources (White 2004) I had noted that the de novo appearance of chloroquine resistance in Plasmodium falciparum was an event of probability of 1 in 10^20. I then wrote that ‘‘for humans to achieve a mutation like this by chance, we would have to wait 100 million times 10 million years’’ (Behe 2007) (because that is the extrapolated time that it would take to produce 10^20 humans). Durrett and Schmidt (2008, p. 1507) retort that my number ‘‘is 5 million times larger than the calculation we have just given’’ using their model (which nonetheless "using their model" gives a prohibitively long waiting time of 216 million years). Their criticism compares apples to oranges. My figure of 10^20 is an empirical statistic from the literature; it is not, as their calculation is, a theoretical estimate from a population genetics model. *******www.discovery****/a/9461 This following calculation by geneticist John Sanford for 'fixing' a beneficial mutation, or for creating a new gene, in humans, gives equally absurd numbers that once again render the Darwinian scenario of humans evolving from apes completely false: Dr. Sanford calculates it would take 12 million years to “fix” a single base pair mutation into a population. He further calculates that to create a gene with 1000 base pairs, it would take 12 million x 1000 or 12 billion years. This is obviously too slow to support the creation of the human genome containing 3 billion base pairs. *******www.detectingtruth****/?p=66 Whale Evolution Vs. Population Genetics - Richard Sternberg PhD. in Evolutionary Biology - video *******www.metacafe****/watch/4165203 Materialists simply do not have the 'beneficial' mutations they need to make evolution work. The following site has numerous quotes, studies and videos which reveal the overwhelmingly negative mutation rate which has been found in life: Mutation Studies, Videos, And Quotes *******docs.google****/Doc?docid=0AYmaSrBPNEmGZGM4ejY3d3pfMjZjZnM5M21mZg An Atheist Interviews Michael Behe About "The Edge Of Evolution" - video *******www.in****/videos/watchvideo-bloggingheads-interview-with-michael-behe-4734623.html Michael Behe's Amazon Blog *******www.amazon****/gp/blog/A3DGRQ0IO7KYQ2/ref=cm_blog_blog/180-3515567-6480413 Michael Behe - bio. and list of peer reviewed papers: *******www.lehigh.edu/~inbios/faculty/behe.html Should Intelligent Design Be Taught as Science? Michael Behe debates Stephen Barr - 2010 - video *******www.isi****/lectures/flvplayer/lectureplayer.aspx?file=v000355_cicero_040710.mp4&dir=mp4/lectures Main page - with audio of debate *******www.isi****/lectures/lectures.aspx?SBy=lecture&SFor=18fdfd28-e682-421f-9acf-2940402af8e3 Peer-Reviewed & Peer-Edited Scientific Publications Supporting the Theory of Intelligent Design (Annotated) - updated regularly *******www.discovery****/a/2640 The DNA Code - Solid Scientific Proof Of Intelligent Design - Perry Marshall - video *******www.metacafe****/watch/4060532/the_dna_code_solid_scientific_proof_of_intelligent_design_perry_marshall/ The current materialistic argument essentially appears to be like this: Premise One: No materialistic cause of specified complex information is known. Conclusion: Therefore, it must arise from some unknown materialistic cause. On the other hand, Stephen Meyer describes the intelligent design argument as follows: “Premise One: Despite a thorough search, no material causes have been discovered that demonstrate the power to produce large amounts of specified information. “Premise Two: Intelligent causes have demonstrated the power to produce large amounts of specified information. “Conclusion: Intelligent design constitutes the best, most causally adequate, explanation for the information in the cell.” Stephen Meyer - Extreme Rarity of Functional Proteins And Higher Level Information For Body Plans - video *******www.metacafe****/watch/4050681 This following video is a bit more clear for explaining exactly why mutations to the DNA do not control Body Plan morphogenesis, since the mutations are the ‘bottom rung of the ladder’ as far as the 'higher levels of the layered information’ of the cell are concerned: Stephen Meyer on Craig Venter, Complexity Of The Cell & Layered Information *******www.metacafe****/watch/4798685 Intelligent Design - The Anthropic Hypothesis *******lettherebelight-77.blogspot****/2009/10/intelligent-design-anthropic-hypothesis_19.html
11 Oct 2010
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